By I. Hertman (auth.), Abraham Fisher, Israel Hanin, Chaim Lachman (eds.)
This booklet is the result of a global convention, held on the Aviya Sonesta lodge in Eilat, Israel, on March 24-27, 1985. This was once the thirtieth in a chain of the yearly OHOLO meetings, backed by means of the Israel Institute for organic examine. members during this convention consisted of scientists from 4 teenager nations; represented a wide spectrum of analysis pursuits; and integrated a well-balanced illustration from academia, scientific associations and pharmaceutical undefined. The e-book contains talks, poster periods, and a entire basic dialogue, all from the court cases of this convention. within the curiosity of assuring a speedy ebook of the unconventional infor mation reviewed, this publication has been ready in camera-ready structure. we're cognizant of the truth that a number of typographical error might exist within the textual content, and that there are adaptations fashionable and typeface of the var ious chapters. This, we felt, used to be an affordable compromise for the sake of pace and effective transmission of worthy clinical info. No physique is ideal . •••• The good fortune of the convention, and for that reason the standard of this booklet are caused by the broad efforts of a giant variety of super able participants. those comprise contributors of the medical Organizing Committee, and the Technical employees of the OHOLO association. in fact, the convention couldn't have succeeded with no the top of the range of the displays, and the enthusiastic participation of the contributing scientists.
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Extra resources for Alzheimer’s and Parkinson’s Disease: Strategies for Research and Development
One possibility is that etiological factors such as an infectious agent might alter the biology of microglia/pericytes such that they become "producer cells" in which amyloidogenic protein arise as a new protein not normally expressed or as an overproduction of a native protein normally present in the ce 11. A second opt ion is that the amyloidogenic protein is derived from either the blood stream or elsewhere in the brain. In this case the microglia behave as "processor" cells and alter the protein(s) in such a way that it now polymerizes into fibers that are deposited locally.
10:122-126 (1981) • Whitehouse. J •• Price. L •• Struble. G •• Clark. W •• Coyle. T •• and DeLong. R •• Alzheimer's disease and senile dementia: Loss of neurons in the basal forebrain. Science 215:1237-1239 (1982). Wisniewski. M •• Bruce. E •• and Fraser. -H .. Infectious etiology of neuritic (senile) plaques in mice. Science. 190:1108-1110 (1975). Wisniewski. M •• Iqbal. R •• Aluminum-induced neurofibrillary changes: Its relationship to senile dementia of the Alzheimer's type. Neurotoxicology 1:121-124 (1980).
Eds •• Volume I. Biomedical Issues, Springer-Verlag, New York, pp. 231-243 (1985). Wisniewski. M •• Merz. A.. and Iqbal. , Ultrastructure of paired 35 helical filaments of Alzheimer's neurofibrillary tangle. J. Neuropath. Exp. Neurol. 43:643-656 (1984). Wisniewski. M •• Moretz. C •• and Lossinsky. S •• Evidence for induction of localized amyloid deposits and neuritic plaques by an infectious agent. Ann. Neurol. 10:517-522 (1981c). Wisniewski. M •• Narang. K •• and Terry. D •• Neurofibrillary tangles of paired helical filaments.